Abstract
Recent research suggests that hyperhomo-cysteinemia is a significant risk factor for thrombosis. The mechanism for this is not completely understood. However, studies have shown that high intakes of folic acid, vitamin B12, and vitamin B6 can lower homocysteine levels. This case study describes a patient with a 7-year history of postprandial pain who was diagnosed with mesenteric ischemia. She was found to have moderate hyperhomocysteinemia and was subsequently treated with B vitamins. Unable to tolerate enteral nutrition, the patient was maintained on- parenteral nutrition before surgery. After resection of ischemic, strictured gut, her parenteral nutrition was discontinued and she gained weight on an oral diet. Her serum homocysteine level dropped to low-normal, therefore, vitamin B supplementation continues. Since surgery, the patient has been without symptoms of chronic ischemia.
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