AlarieY., KaneL., and BarrowC. (1980). Sensory irritation: The use of an animal model to establish acceptable exposure to airborne chemical irritants. In: Toxicology: Principles and Practices, Vol. 1 (ReevesA.L., ed.). John Wiley, New York. pp. 48–91.
2.
AllisonD.J. and PowisD.A. (1971). Adrenal catecholamine secretion during stimulation of the nasal mucous membrane in the rabbit.J. Physiol.217:327–339.
3.
BarnesP.J. (1987). Neuropeptides in the lung: Localization, function and pathophysiologic implication.J. Allergy Clin. Immunol.79:285–295.
BascomR., ChinC-Y., LevittR.C., and KleebergerS.R. (1992). Interstrain differences in the acute response to tobacco smoke.Am. Rev. Respir. Dis. (in press).
6.
BondJ.A. (1986). Bioactivation and biotransformation of xenobiotics in rat nasal tissue. In: Toxicology of the Nasal Passages (BarrowC.S., ed.). Hemisphere Publishing Corporation, Washington, pp. 249–261.
7.
BritteboE.B., CastonguayA., RafterJ.J., KowalskiB., AhlmanM., and BrandtI. (1986). Metabolism of xenobiotics and steroid hormones in the nasal mucosa. In: Toxicology of the Nasal Passages (BarrowC.S., ed.). Hemisphere Publishing Corporation, Washington, pp. 211–234.
8.
CENTERS FOR DISEASE CONTROL (CDC). (1989). Reducing the Health Consequences of Smoking: 25 Years of Progress. A Report of the Surgeon General. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control, Center for Chronic Disease Prevention and Health Promotion, Office on Smoking and Health, Atlanta.
9.
ChurchillL., ChiltonF.H., ResauJ.H., BascomR., HubbardW.C., and ProudD. (1989). Cyclooxygenase metabolism of endogenous arachidonic acid by cultured human tracheal epithelial cells.Am. Rev. Respir. Dis.140:449–459.
10.
CoxG., OhtoshiT., GauldieJ., DenburgJ., and JordanaM. (1990). Human bronchial epithelial cells cause differentiation of HL-60 cells and prolong the in vitro survival of eosinophils.Am. Rev. Respir. Dis.141:A108.
11.
DahlA.R. and HadleyW.M. (1991). Nasal cavity enzymes involved in xenobiotic metabolism: Effects on the toxicity of inhalants.Crit. Rev. Toxicol.21:345–372.
12.
DoupnikC.A. and LeikaufG.D. (1990). Acrolein stimulates eicosanoid release from bovine airway epithelial cells.Am. J. Physiol.259:L222–229.
13.
DusserD.J., DjokicT.D., BorsonD.B., and NadelJ.A. (1989). Cigarette smoke induces bronchoconstrictor hyperresponsiveness to substance P and inactivates airway neutral endopeptidase in the guinea pig.Possible role of free radicals. J. Clin. Invest.84:900–906.
14.
GrahamD.E. and KorenH.S. (1990). Biomarkers of inflammation in ozone-exposed humans.Comparison of the nasal and bronchoalveolar lavage. Am. Rev. Respir. Dis.142:152–156.
15.
HarkemaJ.R. (1990). Comparative pathology of the nasal mucosa in laboratory animals exposed to inhaled irritants.Environ. Health Perspect.85:231–238.
16.
KazazianH.H.Jr., (1976). A geneticist's view of lung disease.Am. Rev. Respir. Dis.113:261–266.
17.
KelleyJ. (1990). State of the art: Cytokines of the lung.Am. Rev. Respir. Dis.141:765–788.
18.
KorenH.S., GrahamD.E., and DevlinR.B. (1992). Exposure of humans to volatile organic compounds (VOC) results in inflammation in the nasal passages. Arch. Environ. Health (in press).
19.
LaitinenL.A., HeinoM., LaitinenA., and HaahtelaT. (1985). Damage of the airway epithelium and bronchial reactivity in patients with asthma.Am. Rev. Respir. Dis.131:599–606.
20.
LEIKAUFG.D., DRISCOLLK.E. and WeyH.E. (1988). Ozone-induced augmentation of eicosanoid metabolism in epithelial cells from bovine trachea.Am. Rev. Respir. Dis.137:435–442.
21.
LEVITTR.C. and MITZNERW. (1988). Expression of airway hyperreactivity to acetylcholine as a simple autosomal recessive trait in mice.FASEB J.2:2605–2608.
22.
LEVITTR.C. and MITZNERW. (1989). Autosomal recessive inheritance of airway hyperreactivity to 5-hydroxytryptamine. J. Appl. Physiol.67:1, 125.
23.
LUNDBERGJ.M. and SARIAA. (1983). Capsaicin-induced desensitization of airway mucosa to cigarette smoke, mechanical and chemical irritants.Nature302:251–253.
24.
LUNDBERGJ.M., LUNDBLADL., SARIAA., and ANGGARDA. (1984). Inhibition of cigarette smoke induced oedema in the nasal mucosa by capsaicin pretreatment and a substance P antagonist.Naunyn-Schmiedeberg's Arch. Pharmacol.326:181–185.
25.
LUNDBLADL., HuaX-Y., and LUNDBERGJ.M. (1984). Mechanisms for reflexive hypertension induced by local application of capsaicin and nicotine to the nasal mucosa.Acta Physiol. Scand.121:277–282.
26.
MATTOLIS., MIANTES., CALABROF., MEZZETTIM., FASOLIA., and ALLEGRAL. (1990). Bronchial epithelial cells exposed to isocyanates potentiate activation and proliferation of T-cells.Am. J. Physiol.259:L320–L327.
27.
NIELSENG.D. (1991). Mechanisms of activation of the sensory irritant receptor by airborne chemicals.CRC Crit. Rev. Toxicol.21:183–208.
28.
SHIMC. and WILLIAMSM.H. (1986). Effect of odors in asthma.Amer. J. Med.80:18–22.
29.
SPEERF. (1968). Tobacco and the non-smoker.A study of subjective symptoms. Arch. Environ. Health16:443–446.