Ahlgren, L. and Mattsson, S. (1979). An X-ray fluorescence technique for in vivo determination of lead concentration in a bone matrix. Physi. Med. Biol . 24: 136-145.
2.
Araki, S., Yokoyama, K., Aono, H., and Murata, K. (1986). Psychologic performance in relation to central and peripheral nerve conduction in workers exposed to lead, zinc, and copper . Amer. J. Industr. Med. 9:535-542.
3.
Araki, S. and Honma, T. (1982). Relationships between lead absorption and peripheral nerve conduction velocities in lead workers. Brit. J. Industr. Med. 39:157-160.
4.
Arnvig, E., Grandjean, P., and Beckmann, J. (1980). Neurotoxic effects of heavy lead exposure determined with psychological tests. Tox. Ltrs. 5:399-404.
5.
Assennato, G., Paci, G., Baser, M.E., Molinini, R., Candela, R.B., Altamura, B.M., and Giogino, R. (1986). Sperm count suppression without endocrine dysfunction in lead-exposed men. Arch. Environ. Hlth . 4:387-390.
6.
Baker, E.L., JR., Landrigan, P.J., Barbour, A.G., Cox, D.H., Folland, D.S., Ligo, R.N., and Throckmorton, J. (1979). Occupational lead poisoning in the United States; clinical and biochemical findings related to blood lead levels. Brit. J. Industr. Med. 36:314-322.
7.
Bellinger, D., Leviton, A., Waternaux, C., Needleman, H., and Rabinowitz, M. (1987). Longitudinal analyses of prenatal and postnatal lead exposure and early cognitive development. N. Engl. J. Med. 316:1037-1043.
8.
Bornschein, R.L., Hammond, P.B., Dietrich, K.N., Succop, N., Krafft, K., Clark, S., Berger, O., Pearson, D., and Que Hee, S. (1985). The Cincinnati prospective study of low-level lead exposure and its effects on child development; protocol and status report . Environ. Res. 38:4-18.
9.
Cooper, W.C. and Gaffey, W.R. (1975). Mortality of lead workers. J. Occ. Med. 17:100-107.
10.
Cullen, M.R., Kayne, R.D., and Robins, J.J. (1984). Endocrine and reproductive dysfunction in men associated with occupational inorganic lead intoxication. Arch. Environ. Hlth. 39:431-440.
11.
Dingwall-Fordyce, I. and Lane, R.E. (1963). A follow-up study of lead workers. Brit. J. Industr. Med. 290:313-315.
12.
Feldman, R.G., Haddon, J., Kopito, L., and Schwachman, H. (1973). Altered peripheral nerve conduction velocity, chronic lead intoxication in children. Amer. J. Dis. Child125:39-41.
13.
Fullerton, P.M. (1966). Chronic peripheral neuropathy produced by lead poisoning in guinea pigs. J. Neuropath. Exp. Neurol . 24:214-236.
14.
Goldstein, B., Gibson, J., Henderson, R., Hobbie, J., Landrigan, P.J., Mattison, D., Perera, F., Silbergeld, E., and Wogan, G. (1987). Biological markers in environmental health research . Environ. Hlth. Persp. 74:3-9.
15.
Goyer, R.A. and Rhyne, B. (1973). Pathological effects of lead. Interntl. Rev. Exper. Path. 12:1-77.
16.
Goyer, R.A., Weinberg, C.R., Victery, W.M., and Miller, C.R. (1987). Lead induced nephrotoxicity: Kidney calcium as an indicator of tubular injury. Proceedings of Third International Conference on Nephrotoxicity, Surrey, England, August 1987 (in press).
17.
Hamilton, A. and Hardy, H.L. (1974). Industrial Toxicology. Acton, MA: Publishing Sciences Group.
18.
Haeger-Aronsen, B., Abdulla, M., and Firstedt, B. (1971). Effect of lead on d-aminolevulinic acid dehydrase activity in red blood cells. Arch. Environ. Hlth . 23:440-445.
19.
Hanninen, H., Hernberg, S., Mantere, P., Vesanto, R., and Jalkanen, M. (1979). Psychological performance of subjects with low exposure to lead. J. Occup. Med. 20:683-689.
20.
Hernberg, S. (1980). Biochemical and clinical effects and responses as indicated by blood lead concentration. In: Singal, R.L., Thomas, J.A., eds. Lead Toxicity. Baltimore: Urban and Schwarzenburg, pp. 367-399.
21.
Hernberg, S., Nikkanene, J., Mellin, G., and Lillius, H. (1971). D-amino-levulinic acid dehydrase activity in red blood cells. Arch. Environ. Hlth. 23:440-445.
22.
Lancranjan, I., Popescu, H.I., Gavenescu, O., Klepsh, I., and Serbanescu, M. (1975). Reproductive ability of workmen occupationally exposed to lead. Arch. Environ. Hlth. 30:396-401.
23.
Letz, R. and Baker, E.L. (1986). Computer-administered neurobehavioral testing in occupational health. Seminars in Occ. Med . 1:197-203.
24.
Mahaffey, K.R. (1985). Health Consequences of Dietary and Environmental Exposure to Lead. Amsterdam: Elsevier North Holland Biomedical Press .
25.
Major, R.H. (1939). Classic descriptions of disease with biographical sketches of the authors , 2nded., Baltimore and Springfield, Illinois: Charles C. Thomas .
26.
Malcolm, D. and Barnett, H.A.R. (1982). A mortality study of lead workers, 1925-1976. Brit. J. Industr. Med. 39:402-404.
27.
Mcmichael, A.J., Baghurst, P.A., Wigg, N.R., Vimpani, G.V., Robertson, E.F., AND Roberts, R.J. (1988). Port Pirie cohort study: Environmental exposure to lead and children's abilities at the age of four years. N. Engl. J. Med. 319:468-475.
28.
Mcmichael, A.J. and Johnson, H.M. (1982). Long-term mortality profile of heavily-exposed lead smelter workers. J. Occup. Med. 24:375-378.
29.
Nordberg, G.F. (1976). Effects and dose-response relationships of toxic metals. In: Nordberg, G.F., ed. Effects and Dose-Response Relationships of Toxic Metals. Amsterdam: Elsevier Scientific Publishing Co.
30.
Olivier, SIR. T. (1914). Lead Poisoning: from the Industrial, Medical , and Social Points of View. Lectures delivered at the Royal Institute of Public Health, New York: Hoeber.
31.
Piomelli, S. (1973). A micromethod for free erythrocyte porphyrins: the FEP test. J. Lab. Clin. Med. 81:932-940.
32.
Pirkle, J.L., Schwartz, J., Landis, R., and Harlan, W.R. (1985). The relationship between blood lead levels and blood pressure and its cardiovascular risk implications. Amer. J. Epidemiol. 121:246-258.
33.
Ramazzini, B. (1913). De Morbis Artificum Diatriba. Trans. Wright, W.C. Chicago: University of Chicago Press.
34.
Rom, W.N. (1980). Effects of lead on reproduction. In: Infante, P.F., Legator, M.S. eds. Proceedings of a Workshop on Methodology for Assessing Reproductive Hazards in the Workplace. Washington, DC: National Institute for Occupational Safety and Health.
35.
Rosen, J.F., Chessney, R.W., Hamstra, A., Deluca, H.F., and Mahaffey, K.R. (1980). Reduction in 1,2,5-dihydroxyvitamin D in children with increased lead absorption. N. Engl. J. Med . 302:1128-1131.
36.
Selevan, S.G., Landrigan, P.J., Stern, F.B., and Jones, J.H. (1985). Mortality of lead smelter workers. Amer. J. Epidemiol. 122:673-683.
37.
Seppäläinen, A.M. and Hernberg, S. (1980). Subclinical lead neuropathy. Amer. J. Industr. Med. 1:413-420.
38.
Seppäläinen, A.M., Hernberg, S., and Kock, B. (1979). Relationship between blood lead levels and nerve conduction velocities. Neurotoxicology1:313-332.
39.
Seppalainen, A.M., Tola, S., Hernberg, S., and Kock, B. (1975). Subclinical neuropathy at "safe" levels of lead exposure. Arch. Environ. Hlth. 30:180.
40.
Seppalainen, A.M., Hernberg, S., Vesanto, R., and Kock, B. (1983). Early neurotoxic effects of lead exposure: a prospective study. Neurotoxicology4:181-192.
41.
Somervaille, L.J., Chettle, D.R., Scott, M.C., Tennant, D.R., Mckiernan, M.J., Skilbeck, A., and Trethowan, W.N. (1988). In vivo tibia lead measurements as an index of cumulative exposure in occupationally exposed subjects. Brit. J. Industr. Med. 45:174-181.
42.
Valciukas, J.A., Lilis, R., Fischbein, A., Selikoff, I.J., Eisinger, J., and Blumberg, W. (1978). Central nervous system dysfunction due to lead exposure. Science201:465-467.
43.
Victery, W., Vander, A.J., and Shulak, A.M. (1982). Lead, hypertension, and the renin-angio-tension system in rats. J. Lab. Clin. Med. 99:354-362.
44.
Waldron, H.A. and Stofen, D. (1974). Subclinical Lead Poisoning. London: Academic Press.
45.
Wielopolski, L., Ellis, K.J., Vaswani, A.N., Cohn, S.H., Greenberg, A., Pushett, J.B., Parkinson, D.K., Fetterolf, D.E., and Landrigan, P.J. (1986). In vivo bone lead measurements: a rapid monitoring method for cumulative lead exposure. Amer. J. Industr. Med. 9:221-226.