Restricted accessResearch articleFirst published online 1985-1
Erythrocyte Glucose-6-Phosphate Dehydrogenase Deficiency Does Not Pose an Increased Risk for Black Americans Exposed To Oxidant Gases in the Workplace or General Environment
Beutler, E., Dern, R.J., Flanagan, C.L. and Alving, A.S. (1955). The hemolytic effect of primaquine. VII. Biochemical studies of drug sensitive erythrocytes. J. Lab. Clin. Med.45:286-295.
2.
Buckley, R.D., Hackney, J.D., Clark, K. and Posin, C. (1975). Ozone and human blood. Arch. Environ. Health30:40-43.
3.
Calabrese, E.J., Kojola, N.H. and Carnow, B.W. (1977). Ozone: a possible cause of hemoloytic anemia in glucose-6-phosphate dehydrogenase deficient individuals. J. Tox. Environ. Health2:709-712.
4.
Calabrese, E.J., Moore, G. and Brown, R. (1979). Effects of environmental oxidant stressors on individuals with a G-6-PD deficiency with particular reference to an animal model. Environ. Health Persp.29:49-55.
5.
Dern, R.J., Beutler, E. and Alving, A. (1955). The hemolytic effect of primaquine V. Primaquine sensitivity as a manifestation of a multiple drug sensitivity. J. Lab. Clin. Med.45:30-39.
6.
Goldstein, B.D., Pearson, B., Lodi, C., Buckley, R. and Balchum, O. (1968). The effect of ozone on mouse blood in vivo. Arch. Environ. Health1:648-650.
Piomelli, S., Corash, L.M., Davensport, D.D., Miraglia, J. and Amorosi, E.L. (1968). In vivo lability of glucose-6-phosphate dehydrogenase in GdA and GdMediterranean Deficiency. J. Clin. Invest47:940-948.
11.
Zinkham, W.H., Lenhard, R.E. and Childs, B. (1958). A deficiency of Glucose-6-phosphate dehydrogenase activity in erythrocyte from patients with favism. Bull. Johns Hopkins Hosp.102:169-175.