BoogaertsM. A.YamadaO.JacobH. S.MoldowC. F.1982. Enhancement of granulocyte-endothelial cell adherence and granulocyte-induced cytotoxicity by platelet release products. Proc. Natl. Acad. Sci U.S.A.;79: 7019–7023.
2.
De ServiS.RicevutiG.MussiniA.MazzoneA.RondanelliR.PomaE.SpecchiaG.MontemartiniC.1988. Neutrophils function in coronary artery disease. Eur. Heart J.;9: 249.
3.
EnglerR.CovellJ. W.1987. Granulocytes cause reperfusion ventricular dysfunction after 15 -minute ischemia in the dog. Circulation Research; 61: 20–28.
4.
FormanM. B.PuettD. W.VirmaniR.1989. Endothelial and myocardial injury during ischemia and reperfusion: Pathogenesis and therapeutic implications. J. Am. Coll. Cardiol;13: 450–459.
HarlanJ. M.1987. Consequence of leukocyte-vessel wall interactions in inflammatory and immune reactions. Seminars Thrombosis and Hemostasis;13: 434–444.
7.
HenningB.ChowC. K.1988. Lipid peroxidation and endothelial cell injury: Implication in athetosclerosis. Free Radical Biology & Medicine: 4: 99–106.
8.
HensonP. M.JohnstonR. B.Jr, 1987. Tissue injury in inflammation: Oxidants, proteinases and cationic proteins. J. Clin. Invest.;79: 669–674.
9.
IngrahamL. M.CoatesT. D.AllenJ. M.HigginsC. P.BaehnerR. L.BoxerL. A.1982. Metabolic, membrane, and functional responses of human polymorphonuclear leukocytes to platelet-activating factor. Blood;59: 1259–1265.
10.
LeRoyE. C.AgerA.GordonJ. L.1984. Effects of neutrophil, elastase and other proteases on porcine aortic endothelial prostaglandin I2 production, adenine nucleotide release, and response to vasoactive agents. J. Clin. Invest.;74: 1003–1010.
11.
MalechH. L.GallinJ. I.1987. Neutrophils in human disease. N. Engl. J. Med.;317: 687–694.
12.
MayrowitzH.WiedemanM.TumaR.1977. Factors influencing leukocyte adherence in microvessels. Thromb Haemost;38: 823–831.
13.
MehtaJ. L.NicholsW. W.MehtaP.1988. Neutrophils as potential participants in acute myocardial ischemia: Relevance to reperfusion. J. Am. Cardiol;11: 1309–1316.
14.
MullaneK. M.WestlinW.KraemerR.1988. Activated neutrophils release mediators that may contribute to myocardial injury and dysfunction associated with ischemia and reperfusion. Ann. N. Y. Acad. Sci.;524: 103–121.
15.
RicevutiG.MazzoneA.1989. The neutrophils revisited. Inflammation;13: 475–483.
16.
RicevutiG.MazzoneA.De ServiS.SpecchiaG.FratinoP.1989. New trends in coronary artery disease: The role of granulocyte activation. Atherosclerosis, in press.
WeissS. J.1989. Tissue destruction by neutrophils. N. Engl. J. Med.;320: 365–376.
19.
WeissS. J.YoungJ.Lo BuglioA. F.SlivkaA.NimehN. F.1981. Role of hydrogen peroxide in neutrophil-mediated destruction of cultured endothelial cells. J. Clin. Invest.;68: 714–721.
20.
WeissmannG.SmolenJ. E.KorchakH. M.1980. Release of inflammatory mediators from stimulated neutrophils. N. Engl. J. Med.;303: 27–34.
21.
WernsS. W.LucchesiB. R.1988. Leukocytes, oxygen radicals, and myocardial injury due to ischemia and reperfusion. Free Radical Biology & Medicine;4: 31–37.
