Abstract
Background: The effect on joint stability of repair of type II superior labrum and biceps anchor lesions is unknown.
Hypothesis: Increased translations of the glenohumeral joint after a simulated type II lesion will be reduced after the lesion is repaired.
Study Design: Controlled laboratory study.
Methods: A robotic/universal force-moment testing system was used to simulate load-and-shift and apprehension tests on eight cadaveric shoulders to determine joint kinematics of the shoulder after venting, creation of a type II lesion, and repair of the lesion.
Results: At 30° of abduction, anterior translation of the vented joint in response to an anterior load was 18.7 ± 8.5 mm and was significantly increased to 26.2 ± 6.5 mm after simulation of a type II lesion. Repair did not restore anterior translation (23.9 ± 8.6 mm) to that of the vented joint. The inferior translation that also occurred during application of an anterior load was 3.8 ± 4.0 mm in the vented joint and increased significantly to 8.5 ± 5.4 mm with a simulated type II lesion. After repair, the inferior translation decreased significantly to 6.7 ± 5.3 mm.
Conclusions: Repair of a type II lesion only partially restored glenohumeral translations to that of the vented joint.
Clinical Relevance: Surgical techniques including improved repair of passive stabilizers injured in the type II lesion should be considered.
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