BuehlerE.V. (1965). Delayed contact hypersensitivity in the guinea pig. Archives of Dermatology91, 171–177.
2.
MagnussonB., & KligmanA.M. (1970). Allergic Contact Dermatitis in the Guinea Pig: Identification of Contact Allergens, 141 pp. Springfield, IL, USA: Charles C Thomas.
3.
KimberI., DearmanR.J., BasketterD.A., RyanC.A., & GerberickG.F. (2002). The local lymph node assay: past, present and future. Contact Dermatitis47, 315–328.
4.
KimberI., PichowskiJ.S., BasketterD.A., & DearmanR.J. (1999). Immune responses to contact allergens: novel approaches to hazard evaluation. Toxicology Letters106, 237–246.
5.
KimberI., PichowskiJ.S., BettsC.J., CumberbatchM., BasketterD.A., & DearmanR.J. (2001). Alternative approaches to the identification and characterization of chemical allergens. Toxicology in Vitro15, 307–312.
6.
RyanC.A., HuletteB.C., & GerberickG.F. (2001). Approaches for the development of cell-based in vitro methods for contact sensitisation. Toxicology in Vitro15, 43–55.
7.
KimberI., CumberbatchM., BettsC.J., & DearmanR.J. (2004). Dendritic cells and skin sensitisation hazard assessment. Toxicology in Vitro18, 195–202.
8.
GrabbeS., & SchwarzT. (1998). Immunoregulatory mechanisms involved in the elicitation of allergic contact hypersensitivity. Immunology Today19, 37–44.
SebastianiS., AlbanesiC., De PitaO., PudduP., CavaniA., & GirolomoniG. (2002). The role of chemokines in allergic contact dermatitis. Archives of Dermatological Research293, 552–559.
12.
DearmanR.J., & KimberI. (2003). Factors influencing the induction phase of skin sensitisation. Americal Journal of Contact Dermatitis14, 188–194.
13.
FriedmannP.S. (1981). The immunobiology of Langerhans cells. Immunology Today2, 124–128.
14.
KimberI., & CumberbatchM. (1992). Dendritic cells and cutaneous immune responses to chemical allergens. Toxicology and Applied Pharmacology117, 137–146.
15.
KimberI., DearmanR.J., CumberbatchM., & HubyR.J.D. (1998). Langerhans cells and chemical allergy. Current Opinion in Immunology10, 614–619.
16.
KimberI., CumberbatchM., DearmanR.J., BhushanM., & GriffithsC.E.M. (2000). Cytokines and chemokines in the initiation and regulation of epidermal Langerhans cell mobilization. British Journal of Dermatology142, 401–412.
17.
MaurerD., & StinglG. (2001). Lagerhans cells. In Dendritic Cells: Biology and Clinical Applications (ed. LotzeM.T., & ThomsonA.W.), 2nd edn, pp. 35–50. San Diego, CA, USA: Academic Press.
CumberbatchM., DearmanR.J., GriffithsC.E.M., & KimberI. (2003). Epidermal Langerhans cell migration and sensitisation to chemical allergens. Acta Pathologica, Microbiologica et Immunologica Scandinavica111, 797–804.
20.
KimberI., CumberbatchM., DearmanR.J., & GriffithsC.E.M. (2002). Danger signals and skin sensitisation. British Journal of Dermatology147, 613–614.
21.
EnkA.H., & KatzS.I. (1992). Early molecular events in the induction phase of contact sensitivity. Proceedings of the National Academy of Sciences USA89, 1398–1402.
22.
de SilvaO., BasketterD.A., BarrattM.D., CorsiniE., CroninM.T.D., DasP.K., DegwertJ., EnkA., GarrigueJ.L., HauserC., KimberI., LepoittevinJ-P., PeguetJ., & PonecM. (1996). Alternative methods for skin sensitisation testing. The report and recommendations of ECVAM workshop 19. ATLA24, 683–705.
23.
EnkA.H., AngeloniV.L., UdeyM.C., & KatzS.I. (1993). An essential role of Langerhans cell-derived IL-1β in the initiation of primary immune responses in skin. Journal of Immunology150, 3698–3704.
24.
ShornickL.P., De TogniP., MariathasanS., GoellnerJ., Strauss-SchoenbergerJ., KarrR.W., FergusonT.A., & ChaplinD.D. (1996). Mice deficient in IL-1β manifest impaired contact hypersensitivity to trinitrochlorobenzene. Journal of Experimental Medicine183, 1427–1436.
25.
CumberbatchM., DearmanR.J., & KimberI. (1997). Langerhans cells require signals from both tumour necrosis factor-α and interleukin-1β for migration. Immunology92, 388–395.
26.
ReutterK., JagerD., DegwertJ., & HoppeU. (1997). In vitro model for contact sensitisation. II. Induction of IL-1β mRNA in human blood-derived dendritic cells by contact sensitisers. Toxicology in Vitro11, 619–626.
27.
PichowskiJ.S., CumberbatchM., DearmanR.J., BasketterD.A., & KimberI. (2000). Investigation of induced changes in interleukin 1β mRNA expression by cultured human dendritic cells as an in vitro approach to skin sensitisation testing. Toxicology in Vitro14, 351–360.
28.
PichowskiJ.S., CumberbatchM., DearmanR.J., BasketterD.A., & KimberI. (2001). Allergen-induced changes in interleukin 1 beta (IL-1 beta) mRNA expression by human blood-derived dendritic cells: inter-individual differences and relevance for sensitisation testing. Journal of Applied Toxicology21, 115–121.
29.
De SmedtA.C.A., Van Den HeuvelR.L., Van TendelooV.F.I., BernemanZ.N., SchoetersG.E.R., WeberE., & TuschlH. (2002). Phenotypic alterations and IL-1β production in CD34+ progenitor- and monocyte-derived dendritic cells after exposure to allergens: a comparative analysis. Archives of Dermatological Research294, 109–116.
30.
StrunkD., RappersbergerK., EggerC., StroblH., KrömerE., ElbeA., MaurerD., & StinglG. (1996). Generation of human dendritic cells/Langerhans cells from circulating CD34+ hematopoietic progenitor cells. Blood87, 1292–1302.
31.
Santiago-SchwarzF., BeliosE., DiamondB., & CarsonS.E. (1992). TNF in combination with GM-CSF enhances the differentiation of neonatal cord blood cells into dendritic cells and macrophages. Journal of Leucocyte Biology52, 274–281.
32.
CauxC., VanbervlietB., MassacrierC., Dezutter-DambuyantC., de Saint-VisB., JacquetC., YonedaK., ImamuraS., SchmittD., & BanchereauJ. (1996). CD34+ hematopoietic progenitors from human cord blood differentiate along two independent dendritic cell pathways in response to GM-CSF+TNF-α. Journal of Experimental Medicine184, 695–706.
33.
ArrighiJ.F., HauserC., ChapuisB., ZublerR., & KindlerV. (1999). Long-term culture of human CD34+ progenitors from FLT3-ligand, thrombopoietin, and stem cell factor induces extensive amplification of a CD34-CD14– and a CD34-CD14+ dendritic cell precursor. Blood93, 2244–2252.
34.
CurtiA., FogliM., RattaM., TuraS., & LemoliR.M. (2001). Stem cell factor and FLT3-ligand are strictly required to sustain the long-term expansion of primitive CD34+DR– dendritic cell precursors. Journal of Immunology166, 848–854.
35.
JaksitsS., KriehuberE., CharbonnierA.S., RappersbergerK., StinglG., & MaurerD. (1999). CD34+ cell-derived CD14+ precursor cells develop into Langerhans cells in a TGF-β1-dependent manner. Journal of Immunology163, 4869–4877.
36.
FerlazzoG., WesaA., WeiW.Z., & GalyA. (1999). Dendritic cells generated from CD34+ progenitor cells or from monocytes differ in their ability to activate antigen-specific CD8+ T cells. Journal of Immunology163, 3597–3604.
37.
SallustoF., & LanzavecchiaA. (1994). Efficient presentation of soluble antigen by cultured human dendritic cells is maintained by granulocyte/macrophage colony stimulating factor plus interleukin-4 and down-regulated by tumor necrosis factor alpha. Journal of Experimental Medicine179, 1109–1118.
38.
SallustoF., CellaM., DanieliC., & LanzavecchiaA. (1995). Dendritic cells use macropinocytosis and the mannose receptor to concentrate macromolecules in the major histocompatibility complex class II compartment: down-regulation by cytokines and bacterial products. Journal of Experimental Medicine182, 389–400.
39.
SnijdersA., KalinskiP., HilkensC.M., & KapsenbergM.L. (1998). High-level IL-12 production by human dendritic cells requires two signals. International Immunology10, 1593–1598.
40.
SantiniS.M., LapentaC., LogozziM., ParlatoS., SpadaM., Di PucchioT., & BelardelliF. (2000). Type I interferon as a powerful adjuvant for monocyte-derived dendritic cell development and activity in vitro and in Hu-PBL-SCID mice. Journal of Experimental Medicine15, 1777–1788.
41.
GeissmannF., ProstC., MonnetJ.P., DyM., BrousseN., & HermineO. (1998). Transforming growth factor beta 1, in the presence of granulocyte/macrophage colony-stimulating factor and interleukin 4, induces differentiation of human peripheral blood monocytes into dendritic Langerhans cells. Journal of Experimental Medicine16, 961–966.
42.
VerrierA.C., SchmittD., & StaquetteM-J. (1999). Fragrance and contact allergens in vitro modulate the HLA-DR and E-cadherin expression on human epidermal Langerhans cells. International Archives of Allergy and Immunology120, 56–62.
43.
RizovaH., CarayonP., BarbierA., LacheretzF., DubertretL., & MichelL. (1999). Contact allergens, but not irritants, alter receptor-mediated endocytosis by human epidermal Langerhans cells. British Journal of Dermatology140, 200–209.
44.
TuschlH., & KovacR. (2001). Langerhans cells and immature dendritic cells as model systems for screening of skin sensitisers. Toxicology in Vitro15, 327–331.
45.
DegwertJ., SteckelF., HoppeU., & KligmanL.H. (1997). In vitro model for contact sensitisation. I. Stimulatory capacities of human blood-derived dendritic cells and their phenotypical alterations in the presence of contact sensitisers. Toxicology in Vitro11, 613–618.
46.
BeckerD., KühnU., LempertzU., EnkA., SalogaJ., & KnopJ. (1997). Flow-cytometric screening for the modulation of receptor-mediated endocytosis in human dendritic cells: implications for the development of an in vitro technique for predictive testing of contact sensitisers. Journal of Immunological Methods203, 171–180.
47.
AibaS., TerunumaA., ManomeH., & TagamiH. (1997). Dendritic cells differently respond to haptens and irritants by their production of cytokines and expression of co-stimulatory molecules. European Journal of Immunology27, 3031–3038.
48.
AibaS., ManomeH., YoshinoY., & TagamiH. (2000). In vitro treatment of human transforming growth factor-1β-treated monocyte-derived dendritic cells with haptens can induce the phenotypic and functional changes similar to epidermal Langerhans cells in the initiation phase of allergic contact sensitivity reaction. Immunology101, 68–75.
49.
CoutantK.D., de Brugerolle de FraissinetteA., CordierA., & UlrichP. (1999). Modulation of the activity of human monocyte-derived dendritic cells by chemical haptens, a metal alleren, and a Staphylococcal superantigen. Toxicological Sciences52, 189–198.
50.
ManomeH., AibaS., & TagamiH. (1999). Simple chemicals can induce maturation and apoptosis of dendritic cells. Immunology98, 481–490.
51.
HuletteB.C., RyanC.A., & GerberickG.F. (2002). Elucidating changes in surface marker expression of dendritic cells following chemical allergen treatment. Toxicology and Applied Pharmacology182, 226–233.
52.
StaquetM.J., SportouchM., JacquetC., SchmittD., GuesnetJ., & Péguet-NavarroJ. (2004). Moderate skin sensitisers can induce phenotypic changes on in vitro generated dendritic cells. Toxicology in Vitro18, 493–500.
53.
AebyP., WyssC., BeckH., GriemP., SchefflerH., & GoebelC. (2004). Characterization of the sensitizing potential of chemicals by in vitro analysis of dendritic cell activation and skin penetration. Journal of Investigative Dermatology122, 1154–1164.
54.
RougierN., RedziniakG., SchmittD., & VincentC. (1998). Evaluation of the capacity of dendritic cells derived from cord blood CD34+ precursors to present haptens to unsensitized autologous T cells in vitro.Journal of Investigative Dermatology110, 348–352.
55.
RougierN., RedziniakG., MouginD., SchmittD., & VincentC. (2000). In vitro evaluation of the sensitisation potential of weak contact allergens using langerhans-like dendritic cells and autologous T cells. Toxicology145, 73–82.
56.
De SmedtA.C.A., Van Den HeuvelR.L., BernemenZ.N., & SchoetersG.E.R. (2001). Modulation of phenotype, cytokine production and stimulatory function of CD34+ cell-derived DC by NiCl2 and SDS. Toxicology in Vitro15, 319–325.
57.
De SmedtA.C.A., Van Den HeuvelR.L., Van TendelooV.F.I., BernemanZ.N., & SchoetersG.E.R.Capacity of CD34+ progenitor-derived dendritic cells to distinguish between sensitisers and irritants. Toxicology Letters, in press.
58.
BoislèveF., Kerdine-RömerS., Rougier-LarzatN., & PallardyM. (2004). Nickel and DNCB induce CCR7 expression on human dendritic cells through different signalling pathways: role of TNF-α and MAPK. Journal of Investigative Dermatology123, 494–502.
59.
BoislèveF., Kerdine-RömerS., & PallardyM. (2004). Implication of the MAPK pathways in the maturation of human dendritic cells induced by nickel and TNF-α. Toxicology206, 233–244.
60.
DietzA.B., BulurP.A., KnutsonG.J., MatasicR., & Vuk-PavlovicS. (2000). Maturation of human monocyte-derived dendritic cells studied by microarray hybridization. Biochemical and Biophysical Research Communications275, 731–738.
61.
Le NaourF., HohenkirkL., GrolleauA., MisekD.E., LescureP., GeigerJ.D., HanashS., & BerettaL. (2001). Profiling changes in gene expression during differentiation and maturation of monocyte-derived dendritic cells using both oligonucleotide microarrays and proteomics. Journal of Biological Chemistry276, 17,920–17,931.
62.
MoschellaF., MaffeiA., CatanzaroR.P., PapadopoulosK.P., SkerrettD., HesdorferC.S., & HarrisP.E. (2001). Transcript profiling of human dendritic cells maturation-induced under defined culture conditions: comparison of the effects of tumour necrosis factor alpha, soluble CD40 ligand trimer and interferon gamma. British Journal of Haematology114, 444–457.
63.
ChenZ., GordonJ.R., ZhangX., & XiangJ. (2002). Analysis of the gene expression profiles of immature versus mature bone-marrow-derived dendritic cells using DNA arrays. Biochemical and Biophysical Research Communications290, 66–72.
64.
LindstedtM., Johansson-LindbomB., & BorrebaeckC.A.K. (2002). Global reprogramming of dendritic cells in response to a concerted action of inflammatory mediators. International Immunology14, 1203–1213.
65.
MessmerD., MessmerB., & ChiorazziN. (2003). The global transcriptional maturation program and stimuli-specific gene expression profiles of human myeloid dendritic cells. International Immunology15, 491–503.
66.
TüreciO., BianH., NestleF.O., RaddrizzaniL., RosinskiJ.A., TassisA., HiltonH., WalsteadM., SahinU., & HammerJ. (2003). Cascades of transcriptional induction during dendritic cell maturation revealed by genome-wide expression analysis. FASEB Journal17, 836–847.
67.
RyanC.A., GildeaL.A., HuletteB.C., DearmanR.J., KimberI., & GerberickG.F. (2004). Gene expression changes in peripheral blood-derived dendritic cells following exposure to a contact allergen. Toxicology Letters150, 301–316.
68.
KühnU., BrandP., WillemsenJ., JonuleitH., EnkA.H., van Brandwijk-PeterhansR., SalogaJ., KnopJ., & BeckerD. (1998). Induction of tyrosine phosphorylation in human MHC Class II-positive antigen-presenting cells by stimulation with contact sensitisers. Journal of Immunology160, 667–673.
69.
ArdeshnaK.M., PizzeyA.R., DevereuxS., & KhwajaA. (2000). The PI3 kinase, p38 SAP kinase, and NF-kappaB signal transduction pathways are involved in the survival and maturation of lipopolysaccharide-stimulated human monocyte-derived dendritic cells. Blood96, 1039–1046.
70.
UnderhillD.M., & OzinskyA. (2002). Toll-like receptors: key mediators of microbe detection. Current Opinion in Immunology14, 103–110.
71.
DunneA., & O'NeillL. A. (2003). The interleukin-1 receptor/Toll-like receptor superfamily: signal transduction during inflammation and host defense. Science's STKE 2003 (171), re3.
72.
AibaS., ManomeH., NakagawaS., MollahZ.U., MizuashiM., OhtaniT., YoshinoY., & TagamiH. (2003). p38 Mitogen-activated protein kinase and extracellular signal-regulated kinases play distinct roles in the activation of dendritic cells by two representative haptens, NiCl2 and 2,4-dinitrochlorobenzene. Journal of Investigative Dermatology120, 390–399.
73.
ArrighiJ. F., RebsamenM., RoussetF., KindlerV., & HauserC. (2001). A critical role for p38 mitogen-activated protein kinase in the maturation of human blood-derived dendritic cells induced by lipopolysaccharide TNF-alpha and contact sensitisers. Journal of Immunology166, 3837–3845.
74.
BennettB.L., SasakiD.T., MurrayB.W., O'LearyE.C., SakataS.T., XuW., LeistenJ.C., MotiwalaA., PierceS., SatohY., BhagwatS.S., ManningA.M., & AndersonD.W. (2001). SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase. Proceedings of the National Academy of Sciences USA98, 13,681–13,686.
75.
LaderachD., CompagnoD., DanosO., VainchenkerW., & GalyA. (2003). RNA interference shows critical requirement for NF-kappa B p50 in the production of IL-12 by human dendritic cells. Journal of Immunology15, 1750–1757.
76.
LuftT., JeffordM., LuetjensP., ToyT., HochreinH., MastermanK.A., MaliszewskiC., ShortmanK., CebonJ., & MaraskovskyE. (2002). Functionally distinct dendritic cell (DC) populations induced by physiologic stimuli: prostaglandin E(2) regulates the migratory capacity of specific DC subsets. Blood100, 1362–1372.
77.
ScandellaE., MenY., GillessenS., ForsterR., & GroettrupM. (2002). Prostaglandin E2 is a key factor for CCR7 surface expression and migration of monocyte-derived dendritic cells. Blood100, 1354–1361.
78.
BourH., PeyronE., GaucherandM., GarrigueJ.L., DesvignesC., KaiserlianD., RevillardJ.P., & NicolasJ.F. (1995). Major histocompatibility complex class I-restricted CD8+ T cells and class II-restricted CD4+ T cells, respectively, mediate and regulate contact sensitivity to dinitrofluorobenzene. European Journal of Immunology25, 3005–3010.
79.
AndersonC., HehrA., RobbinsR., HasanR., AtharM., MukhtarH., & CraigE. (1995). Metabolic requirements for induction of contact hypersensitivity to immunotoxic polyaromatic hydrocarbons. Journal of Immunology155, 3530–3535.
80.
XuH., DilulioN.A., & FairchildR.L. (1996). T cell populations primed by hapten sensitisation in contact sensitivity are distinguished by polarized patterns of cytokine production: interferon gamma-producing (Tc1) effector CD8+ T cells and interleukin (IL) 4/IL-10-producing (Th2) negative regulatory CD4+ T cells. Journal of Experimental Medicine183, 1001–1012.
81.
MoulonC., WildD., DormoyA., & WeltzienH.U. (1998). MHC-dependant and independent activation of human nickel-specific CD8+ cytotoxic T cells from allergic donors. Journal of Investigative Dermatology111, 360–366.
82.
HauserC., & KatzS.I. (1990). Generation and characterization of T-helper cells by primary in vitro sensitisation using Langerhans cells. Immunological Reviews117, 67–84.
83.
MoulonC., Peguet-NavarroJ., CourtellemontP., RedziniakG., & SchmittD. (1993). In vitro primary sensitisation and restimulation of hapten-specific T cells by fresh and cultured epidermal Langerhans’ cells. Immunology80, 373–379.
84.
KrastevaM., Peguet-NavarroJ., MoulonC., CourtellemontP., RedziniakG., & SchmittD. (1996). In vitro primary sensitisation of hapten-specific T cells by cultured human epidermal Langerhans cells: a screening predictive assay for contact sensitisers. Clinical and Experimental Allergy26, 563–570.
85.
StreileinJ.W., & GrammerS.F. (1989). In vitro evidence that Langerhans cells can adopt two functionally distinct forms capable of antigen presentation to T lymphocytes. Journal of Immunology143, 3925–3933.
86.
PuréE., InabaK., & CrowleyM.T. (1990). Antigen processing by epidermal Langerhans cells correlates with the level of biosynthesis of major histocompatibility complex class II molecules and expression of invariant chain. Journal of Experimental Medicine172, 1459–1469.
87.
RomaniN., GrunerS., BrangD., KampgenE., LenzA., TrockenbacherB., KonwalikaG., FritschP.O., SteinmanR.M., & SchulerG. (1994). Proliferating dendritic cell progenitors in human blood. Journal of Experimental Medicine180, 83–93.
88.
CauxC., MassacrierC., Dezutter-DambuyantC., VanbervlietB., JacquetC., SchmittD., & BanchereauJ. (1995). Human dendritic cells Langerhans cells generated in vitro from CD34+ progenitors can prime naïve CD4+ T cells and process soluble antigen. Journal of Immunology155, 5427–5435.
89.
RustemeyerT., De LigterS., Von BlombergB.M., FroschP.J., & ScheperR.J. (1999). Human T lymphocyte priming in vitro by haptenated autologous dendritic cells. Clinical and Experimental Immunology117, 209–216.
90.
DaiR., & StreinleinJ.W. (1998). Naïve, hapten-specific human T lymphocytes are primed in vitro with derivatized blood mononuclear cells. Journal of Investigative Dermatology110, 29–33.
91.
GuironnetG., Dalbiez-GauthierC., RoussetF., SchmittD., & Peguet-NavarroJ. (2000). In vitro human T cell sensitisation to haptens by monocyte-derived dendritic cells. Toxicology in Vitro14, 517–522.
92.
OpdykeD.L.J. (1975). Monograph on fragrance raw material. Food and Cosmetics Toxicology13, 681–923.
93.
De GrootA.C., LiemD.H., NaterJ.P., & van KetelW.G. (1985). Patch tests with fragrance materials and preservatives. Contact Dermatitis12, 87–92.
94.
LarsenW., NakayamaH., LindbergM., FisherT., ElsnerP., BurrowsD., JordanW., ShawS., WilkinsonJ., MarksJ.Jr., SugawaraM., & NethercottJ. (1996). Fragrance contact dermatitis: a worldwide multicenter investigation (Part I). American Journal of Contact Dermatitis7, 77–83.
95.
LarsenW.G. (2002). Fragrance testing in the 21st century. Contact Dermatitis46, 60–61.
96.
JohansenJ.D., AndersenT.F., KjollerM., VeienN., AvnstorpC., AndersenK.E., & MenneT. (1998). Identification of risk products for fragrance contact allergy: a case-referent study based on patient's histories. American Journal of Contact Dermatitis9, 80–86.
97.
GoodwinB.J.F., CrevelR.W.R., & JohnsonA.W. (1981). A comparison of three guinea pig sensitisation procedures for the detection of 19 reported human contact sensitisers. Contact Dermatitis7, 248–258.
98.
MarzulliF.N., & MaibachH.I. (1974). The use of graded concentrations in studying skin sensitisers: experimental contact sensitisation in man. Food and Cosmetics Toxicology12, 219–227.
99.
BasketterD.A., & ScholesE.W. (1992). Comparison of the local lymph node assay with the guinea pig maximization test for the detection of a range of contact allergens. Food and Cosmetics Toxicology30, 65–69.
100.
BasketterD.A., & LidenC. (1992). Further investigation on the prohapten concept: reactions to benzene derivative in man. Contact Dermatitis27, 90–97.
101.
RégnierM., StaquetM.J., SchmittD., & SchmidtR. (1997). Integration of Langerhans cells into a pigmented reconstructed human epidermis. Journal of Investigative Dermatology109, 510–512.
FacyS., FlouretV., RegnierM., & SchmidtR. (2004). Langerhans cells integrated into human recontructed epidermis respond to known sensitisers and ultraviolet exposure. Journal of Investigative Dermatology122, 552–553.
105.
SchemppC.M., DittmarH.C., HummierD., Simon-HaarhausB., Schulte-MontingJ., SchopfE., & SimonJ.C. (2000). Magnesium ions inhibit the antigen-presenting function of human epidermal Langerhans cells in vivo and in vitro. Involvement of ATPase, HLA-DR, B7 molecules, and cytokines. Journal of Investigative Dermatology115, 680–686.
106.
LarsenC.P., SteinmanR.M., Witmer-PackM., HankinsD.F., MorrisP.J., & AustynJ.M. (1990). Migration and maturation of Langerhans cells in skin transplants and explants. Journal of Experimental Medicine172, 1483–1493.
107.
HerouetC., CottinM., LeclaireJ., EnkA., & RoussetF. (2000). Contact sensitisers specifically increase MHC class II expression on immature dendritic cells. In Vitro and Molecular Toxicology13, 113–123.
108.
St LouisD.C., WoodcockJ.B., FransozoG., BlairP.J., CarlsonL.M., MurilloM., WellsM.R., WilliamsA.J., SmootD.S., KaushalS., GrimesJ.L., HarlanD.M., ChuteJ.P., JuneC.H., SiebenlistU., & LeeK.P. (1999). Evidence for distinct intracellular signaling pathways in CD34+ progenitor to dendritic cell differentiation from a human cell line model. Journal of Immunology162, 3237–3248.
109.
AshikagaT., HoyaM., ItagakiH., KatsumuraY., & AibaS. (2002). Evaluation of CD86 expression and MHC class II molecule internalization in THP-1 human monocyte cells as predictive endpoints for contact sensitisers. Toxicology in Vitro16, 711–716.
110.
YoshidaY., SakaguchiH., ItoY., OkudaM., & SuzukiH. (2003). Evaluation of the skin sensitisation potential of chemicals using expression of co-stimulatory molecules, CD54 and CD86, on the naive THP-1 cell line. Toxicology in Vitro17, 221–228.
111.
KossA., LuceroG., & KozinerB. (1996). Granulocyte-colony stimulating factor, granulocyte-macrophage colony stimulating factor and interleukin 4 induce differentiation in the U-937 human monocytic leukemia cell line. Leukemia and Lymphoma22, 163–171.
112.
TennoT., ObergF., NilssonK., & SiegbahnA. (1997). Induction of differentiation in U-937 and NB4 cells is associated with inhibition of tissue factor production. European Journal of Haematology63, 112–119.
113.
OECD (2002). OECD Guidelines for the Testing of Chemicals, No. 429, The Local Lymph Node Assay, 7 pp. Paris, France: Organization for Economic Cooperation and Development.
114.
RyanC.A., CruseL.W., SkinnerR.A., DearmanR.J., KimberI., & GerberickG.F. (2002). Examination of a vehicle for use with water soluble materials in the murine local lymph node assay. Food and Chemical Toxicology40, 1719–1725.
115.
GerberickG.F., RyanC.A., KimberI., DearmanR.J., LeaL.J., & BasketterD.A. (2000). Local lymph node assay validation assessment for regulatory purposes. American Journal of Contact Dermatitis11, 3–18.
116.
NIH (1999). The Murine Local Lymph Node Assay: A Test Method for Assessing the Allergic Contact Dermatitis Potential of Chemicals/Compounds. NIH No. 99-4494. 211 pp. Bethesda, MD, USA: National Institutes of Health.
117.
GerberickG.F., RobinsonM.K., RyanC.A., DearmanR.J., KimberI., BasketterD.A., WrightZ.M., & MarksJ.G. (2001). Contact allergenic potency: correlation of human and local lymph node assay data. American Journal of Contact Dermatitis12, 156–161
118.
SmithC.K., & HotchkissS.A. (2001). Allergic Contact Dermatitis. Chemical and Metabolic Mechanisms, 310 pp. London, UK: Taylor & Francis.
119.
Smith-PeaseC.K., BasketterD.A., & PatlewiczG.Y. (2003). Contact allergy: the role of skin chemistry and metabolism. Clinical and Experimental Dermatology2, 1777–1783.
120.
BasketterD.A., ScholesE.W., EvansC.D., & KimberI. (1992). Divergent responses to sulphanilic acid in the guinea pig maximization test and the local lymph node assay. Contact Dermatitis27, 209–213.
121.
WahlbergJ.E. (1989). Nickel: animal sensitisation assays. Nickel and skin. In Immunobiology and Toxicology (ed. MaibachH.I., & MennéT.), pp. 65–74. Boca Raton, FL, USA: CRC Press.
122.
BasketterD.A., LeaL., CooperK., RyanC.J., GerberickG.F., DearmanR.J., & KimberI. (1999). The identification of metal allergens in the local lymph node assay. American Journal of Contact Dermatitis10, 207–212.
123.
LepoittevinJ-P., BasketterD.A., Dooms-GoossensA., & KarlbergA-T. (1997). Allergic Contact Dermatitis: The Molecular Basis, 186 pp. Heidelberg, Germany: Springer-Verlag.
124.
ThierseH.J., MoulonC., AllespachY., ZimmermannB., DoetzeA., KuppigS., WildD., HerbergF., & WeltzienH.U. (2004). Metal-protein complex-mediated transport and delivery of Ni2+ to TCR/MHC contact sites in nickel-specific human T cell activation. Journal of Immunology172, 1926–1934.
125.
BasketterD.A., Pease SmithC.K., & PatlewiczG.Y. (2003). Contact allergy: the local lymph node assay for the prediction of hazard and risk. Clinical and Experimental Dermatology28, 218–221.
126.
FelterS.P., RyanC.A., BasketterD.A., & GerberickG.F. (2003). Application of the risk assessment paradigm to the induction of allergic contact dermatitis. Regulatory Toxicology and Pharmacology37, 1–10.
127.
DearmanR.J., SmithS., BasketterD.A., & KimberI. (1997). Classification of chemical allergens according to cytokine secretion profiles of murine lymph node cells. Journal of Applied Toxicology17, 53–62.