Frayn KN: Hormonal control of metabolism in trauma and sepsis. Clin Endocrinol (Oxf)24:577-599, 1986
2.
Wolfe RR, Shaw JHF, Durkot MJ: Effect of sepsis on VLDL kinetics: Responses in basal state and during glucose infusion. Am J Physiol248:E732-E740, 1985
3.
Wolfe RR, Shaw JHF: Glucose and FFA kinetics in sepsis: Role of glucagon and sympathetic nervous system activity. Am J Physiol248:E236-E242, 1985
4.
Romanosky AJ , Bagby GJ, Bockman EL, et al: Free fatty acid utilization by skeletal muscle after endotoxin administration. Am J Physiol239:E391-E395, 1980
5.
Bagby GJ, Pekala PH: Lipoprotein lipase in trauma and sepsis. IN Lipoprotein Lipase , Borensztajn J (ed). Evener, Chicago, 1987, p 247-275
6.
Neufeld HA, Pace JGKaminski MV, et al: A probable endocrine basis for the depression of ketone bodies during infectious or inflammatory state in rats. Endocrinology107:596-601, 1980
7.
Romanosky AJ , McGuinness OP, Spitzer JJ: Metabolic clearance rate of ketone bodies in dogs following Escherichia coli endotoxin administration . Circ Shock11:311-318. 1983
8.
Kovach AGB.Kovach E.Sandor P., et al: Metabolic responses to localized ischemia in adipose tissue . J Surg Res20:37-44. 1976
9.
Lang CH, Bagby GJ, Ferguson JL, et al: Cardiac output and redistribution of organ blood flow in hypermetabolic sepsis. Am J Physiol246:R331-R337, 1984
10.
Spitzer JJ: Lipid metabolism in endotoxin shock. Circ Shock1(Suppl):69-79. 1979
11.
Daniel AM, Pierce CHShizgal HM et al: Protein and fat utilization in shock. Surgery84:588-594. 1978
12.
Spitzer JJ, Bechtel AAArcher LT et al: Myocardial substrate utilization in dogs following endotoxin administration. Am J Physiol227:132-136, 1974
13.
Bagby, GJ, Corll CB, Martinez RR: Triacylglycerol kinetics in endotoxic rats with suppressed lipoprotein lipase activity. Am J Physiol253:E59-E64, 1987
14.
Scholl RA, Lang CH, Bagby GJ: Hypertriglyceridemia and its relation to tissue lipoprotein lipase activity in endotoxemic, Escherichia coli bacteremic, and polymicrobial septic rats . J Surg Res37:394-401, 1984
15.
Beutler BA, Milsark IW, Cerami A.: Cachectin/tumor necrosis factor: Production, distribution, and metabolic fate in vivo. J Immunol135:3972-3977, 1985
16.
Tracey KJ, Fong Y., Hesse DG, et al: Anti-cachectin/TNF monoclonal antibodies prevent septic shock during lethal bacteremia. Nature330:662-664, 1987
17.
Price SR, Mizel SB, Pekala PH: Regulation of lipoprotein lipase synthesis and 3T3-L1 adipocyte metabolism by recombinant interleukin 1. Biochim Biophys Acta89:374-381, 1986
18.
Robin AP, Askanazi J., Greenwood MR, et al: Lipoprotein lipase activity in surgical patients: Influence of trauma and infection. Surgery90:401-408, 1981
19.
Robin AP, Nordenstrom J., Askanazi J., et al: Plasma clearance of fat emulsion in trauma and sepsis: Use of a three-stage lipid clearance test. JPEN4:505-510, 1980
20.
Lang CH, Bagby GJ, Bornside GH, et al: Sustained hypermetabolic sepsis in rats: Characterization of the model. J Surg Res35:201-210, 1983
21.
Lang CH, Bagby GJ, Blakesley HL, et al: Glucose kinetics and pyruvate dehydrogenase activity in septic rats treated with dichloroacetate. Circ Shock23:131-141, 1987
22.
Flynn JT: The role of arachidonic acid metabolites in endotoxin shock. II. Involvement of prostanoids and thromboxanes. IN Handbook of Endotoxin, vol 2, Pathophysiology of Endotoxin, Hinshaw LB (ed). Elsevier, New York, 1985, p 237-285
23.
Lefer AM: Leukotrienes as mediators of ischemia and shock. Biochem Pharmacol35:123-127, 1986
24.
Lang CH, Bagby GJ, Blakesley HL, et al: Inhibition of eicosanoid production by BW755C does not attenuate sepsis-induced alterations in glucose kinetics. Circ Shock22:105-113, 1987
25.
Lang CH, Bagby GJ, Blakesley HL, et al: Fever is not responsible for the elevated glucose kinetics in sepsis. Proc Soc Exp Biol Med185:455-461, 1987
26.
Gelfand RA, Matthews DE, Bier DM, et al: Role of counterregulatory hormones in the catabolic response to stress. J Clin Invest74:2238-2248, 1984
27.
Hargrove DM , Bagby GJ, Lang CH, et al: Adrenergic blockade does not abolish or prevent elevated glucose turnover in sepsis. Am J Physiol254:E16-E22, 1988
28.
Le J., Vilcek J.: Tumor necrosis factor and interleukin 1: Cytokines with multiple overlapping biological activities. Lab Invest56:234-248, 1987
29.
Dinarello CA : Interleukin-1. Rev Infect Dis6:51-95, 1984
30.
Tracey KJ, Beutler B., Lowry SF, et al: Shock and tissue injury induced by recombinant human cachectin . Science234:470-474, 1986
31.
Tracey KJ, Lowry SF, Fahey TJ, et al: Cachectin/tumor necrosis factor induces lethal shock and stress hormone responses in the dog. Surg Gynecol Obstet164:415-422. 1987
32.
Bagby GJLang CHHargrove DM, et al: Glucose kinetics in rats infused with endotoxin-induced monokines or tumor necrosis factor. Circ Shock24:111-121. 1988
33.
Sokoloff LMReivich M.Kennedy C., et al: The [24C] deoxyglucose method for the measurement of local cerebral glucose utilization: Theory, procedure, and normal values in the conscious and anesthetized albino rat . J Neurochem28:897-916, 1977
34.
Meszaros K., Bagby GJ, Lang CH, et al: Contribution of different organs to increased glucose consumption after endotoxin administration. J Biol Chem262:10965-10970, 1987
35.
Meszaros K., Bagby GJ, Lang CH, et al: Increased uptake and phosphorylation of 2-deoxyglucose by skeletal muscle in endotoxin-treated rats. Am J Physiol253:E33-E39, 1987
36.
Meszaros K. , Lang CH, Bagby GJ, et al: In vivo glucose utilization by individual tissues during nonlethal hypermetabolic sepsis. FA-SEB J, 1988
37.
Westfall MV , Sayeed MM: 3-O-methylglucose transport in soleus muscle of bacteremic rats. Am J Physiol253:R55-R63, 1987
38.
Meszaros K. , Lang CH, Bagby GJ, et al: Tumor necrosis factor increases in vivo glucose utilization of macrophage-rich tissues. Biochem Biophys Res Commun149:1-6, 1987