Abstract
A patient is reported who developed lactic acidosis, hyponatraemia and features of the Wernicke-Korsakoff syndrome following long term excessive ingestion of beer and lager. Intramuscular thiamine caused a rapid correction of the lactic acidosis, and the hyponatraemia was corrected by intravenous therapy. On discharge after six weeks of intramuscular Parentrovite the patient still retained some chronic features of the Wernicke-Korsakoff syndrome.
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References
1.
Perkin
GD
Handler
GE
. Wernicke-Korsakoff syndrome . Br J Hosp Med
1983 ; 30 : 331 –334 .
2.
Whyte
KF
Dunnigan
MC
McIntosh
WB
. Excessive beer consumption and beri-beri . Scott Med J
1982 ; 28 : 288 –291 .
3.
Schwatau
M
Doehn
M
Hw
Bause
. Lacticazidose Bei Thiaminmangel . Klinische Wochen — Schrift
1981 ; 59 : 1267 –1270 .
4.
Cohen
RD
Woods
HF
. Clinical and biochemical aspects of lactic acidosis , Oxford ; Blackwell Scientific Publications , 1976 : 41 , 47 .
5.
Mitchell
DM
Kennedy
PGE
. Hyponatremia and its management . Hosp Update
1979 ; 5 : 755 –771 .
6.
Victor
M
Adams
RD
Collins
GH
. The Wernicke-Korsakoff syndrome . Contemporary neurology series
1971 ; Blackwell Scientific
1971 ; 7 .