AblondiF. B.HaganJ. J.PhilipsM.De RenzoE. C. (1959). Inhibition of plasmin, trypsin and streptokinase-activated fibrinolytic system by epsilon aminocaproic acid. Arch. Biochem., 82, 153
2.
AlkjaersigN.FletcherA. P.SherryS. (1959). Epsilon aminocaproic acid: An inhibitor of plasminogen activation. J. biol. Chem., 234, 832
3.
BangN. U.FletcherA. P.AlkjaersigN.SherryS. (1962). Pathogenesis of the coagulation defect developing during plasma proteolytic (‘fibrinolytic’) states. III Demonstration of abnormal clot structure by electron microscopy. J. clin. Invest. 41, 135
4.
FletcherA. P. (1950). Pathophysiology of thrombolysis, In N.I.H. Conference on thrombolytic agents, p. 148. Edited by RobertsH. R.GeratyJ. D.. Chapel Hill: University of North Carolina Press
5.
FletcherA. P.AlkjaersigN.SherryS. (1959a) Pathogenesis of the hemorrhagic diathesis developing during ‘fibrinolytic’ states: The significance of defective fibrin polymerization. J. clin. Invest., 38, 1005. (Abst.)
6.
FletcherA. P.AlkjaersigN.SherryS. (1959b) The maintenance of a sustained throm: Bolytic state in man I. Induction and effects. J. clin. Invest., 38, 1096
7.
JohnsonA. J.NewmanJ.SchoicketD. P. (1960). Inhibition of pathological spontaneous proteolysis in man by the intravenous infusion of epsilon aminocaproic acid, an inhibitor of plasminogen activation. Clin. Res. Proc., 8, 17
8.
Von KaullaK. N.SwanH. (1958). Clotting deviations in man during cardiac bypass: Fibrinolysis and circulating anticoagulant. J. Thorac. Surg., 36, 519
9.
McNicolG. P.DouglasA. S. (1962). To be published
10.
McNicolG. P.FletcherA. P.AlkjaersigN.SherryS. (1961a). Impairment of hemostasis in the urinary tract: The role of urokinaseJ. Lab. clin. Med., 58, 34
11.
McNicolG. P.FletcherA. P.AlkjaersigN.SherryS. (1961b) The use of epsilon aminocaproic acid, a potent inhibitor of fibrinolytic activity, in the management of postoperative hematuria. J. Urol. (Baltimore), 86, 829
12.
McNicolG. P.FletcherA. P.AlkjaersigN.SherryS. (1962a) Plasma amino acid chromatography with ion exchange resin loaded paper: Assay of epsilon aminocaproic acid. J. Lab. clin., Med., 59, 7
13.
McNicolG. P.FletcherA. P.AlkjaersigN.SherryS. (1962b) The absorption, distribution, and excretion of epsilon aminocaproic acid following oral or intravenous administration to man. J. Lab. clin. Med., 59, 15
14.
NilssonI. M.SjoerdsmaA.WaldenstromJ. (1960). Antifibrinolytic activity and metabolism of epsilon aminocaproic acid. Lancet, 1, 1322
15.
ScheragaH. A. (1958). Thrombin and its interaction with fibrinogen. Ann. N.Y. Acad. Sci., 75, 189
16.
SchneiderC. L. (1959). Etiology of fibrinopenia: Fibrination-defibrination. Ann. N.Y. Acad. Sci., 75, 634
17.
SherryS.FletcherA. P.AlkjaersigN. (1959a) Fibrinolysis and fibrinolytic activity in man. Physiol. Rev., 39, 343
18.
SherryS.FletcherA. P.AlkjaersigN.SawyerW. D. (1959b) Epsilon aminocaproic acid. ‘A potent anti-fibrinolytic agent’. Trans. Ass. Amer. Phycns, 72, 62
19.
SoulierJ. P.MatheyJ.Le BollockA. G.DaumetP.FayetH. (1952). Syndromes hémorragiques mortels avec incoagulabilité totale par défibrination et avec fibrinolyse. I. Au cours des exérèses pulmonaires. Rev. Hémat., 7, 30