MarinkovicT, GarinA, YokotaY, FuYX, RuddleNH, FurtadoGC, LiraSA. 2006. Interaction of mature CD3+CD4+ T cells with dendritic cells triggers the development of tertiary lymphoid structures in the thyroid. J Clin Invest, 116:2622–2632.
3.
RoseNR, MolotchnikoffMF, TwarogFJ. 1973. Factors affecting transfer of experimental autoimmune thyroiditis in rats. Immunology, 24:859–870.
4.
LevesqueMC. 2009. Translational mini-review series on b cell-directed therapies: recent advances in B cell-directed biological therapies for autoimmune disorders. Clin Exp Immunol, 157:198–208.
5.
MartinAP, CoronelEC, SanoG, ChenSC, VassilevaG, Canasto-ChibuqueC, SedgwickJD, FrenettePS, LippM, FurtadoGC, LiraSA. 2004. A novel model for lymphocytic infiltration of the thyroid gland generated by transgenic expression of the CC chemokine CCL21. J Immunol, 173:4791–4798.
6.
KotaniT, AratakeY, HiraiK, FukazawaY, SatoH, OhtakiS. 1995. Apoptosis in thyroid tissue from patients with Hashimoto's thyroiditis. Autoimmunity, 20:231–236.
7.
WangSH, BakerJR. 2007. The role of apoptosis in thyroid autoimmunity. Thyroid, 17:975–979.
8.
HayashiY, TamaiH, FukataS, HirotaY, KatayamaS, KumaK, KumagaiLF, NagatakiS. 1985. A long term clinical, immunological, and histological follow-up study of patients with goitrous chronic lymphocytic thyroiditis. J Clin Endocrinol Metab, 61:1172–1178.
9.
CaturegliP, HejaziM, SuzukiK, DohanO, CarrascoN, KohnLD, RoseNR. 2000. Hypothyroidism in transgenic mice expressing IFN-gamma in the thyroid. Proc Natl Acad Sci U S A, 97:1719–1724.
10.
KimuraH, KimuraM, WestraWH, RoseNR, CaturegliP. 2005. Increased thyroidal fat and goitrous hypothyroidism induced by interferon-gamma. Int J Exp Pathol, 86:97–106.
11.
KimuraHJ, RocchiR, Landek-SalgadoMA, SuzukiK, ChenCY, KimuraM, RoseNR, CaturegliP. 2009. Influence of signal transducer and activator of transcription-1 signaling on thyroid morphology and function. Endocrinology, 150:3409–3416.
12.
KimuraHJ, ChenC, TzouSC, RocchiR, Landek-SalgadoM, SuzukiK, KimuraM, RoseNR. Caturegli P 2009 Immunoproteasome overexpression underlies the pathogenesis of thyroid oncocytes, primary hypothyrodism: studies in humans, mice. PLoS ONE, 4,11:e7857.