SkoviraJ.W., KabadiS.V., WuJ., ZhaoZ., DuBoseJ., RosenthalR., FiskumG., and FadenA.I.Simulated aeromedical evacuation exacerbates experimental brain injury. J. Neurotrauma. Epub ahead of print.
2.
LeitchD.R., and HallenbeckJ.M. (1984). Remote monitoring of neuraxial function in anesthetized dogs in compression chambers. Electroencephalogr. Clin. Neurophysiol., 57, 548–560.
3.
HovdaD.A., LeeS.M., SmithM.L., Von StuckS., BergsneiderM., KellyD., ShalmonE., MartinN., CaronM., MazziottaJ., et al. (1995). The neurochemical and metabolic cascade following brain injury: moving from animal models to man. J. Neurotrauma, 12, 903–906.
4.
BoumaG.J., and MuizelaarJ.P. (1929). Cerebral blood flow, cerebral blood volume, and cerebrovascular reactivity after severe head injury. J. Neurotrauma., 9, Suppl 1, S333–S348.
5.
CoppelJ., HennisP., Gilbert-KawaiE., and GrocottM.P. (2015). The physiological effects of hypobaric hypoxia versus normobaric hypoxia: a systematic review of crossover trials. Extrem. Physiol. Med., 4, 2.
6.
ProctorJ.L., ScutellaD., PanY., VaughanJ., RosenthalR.E., PucheA., and FiskumG. (2015). Hyperoxic resuscitation improves survival but worsens neurologic outcome in a rat polytrauma model of traumatic brain injury plus hemorrhagic shock. J. Trauma Acute Care Surg., 79, Suppl 2, S101–S109.
7.
BlasioleB., BayrH., VagniV.A., Janesko-FeldmanK., CheikhiA., WisniewskiS.R., LongJ.B., AtkinsJ., KaganV., and KochanekP.M. (2013) Effect of hyperoxia on resuscitation of experimental combined traumatic brain injury and hemorrhagic shock in mice. Anesthesiology, 118, 649–663.
8.
VereczkiV., MartinE., RosenthalR.E., HofP.R., HoffmanG.E., and FiskumG. (2006). Normoxic resuscitation after cardiac arrest protects against hippocampal oxidative stress, metabolic dysfunction, and neuronal death. J. Cereb. Blood Flow Metab., 26, 821–835.
9.
KochanekP.M., and BayirH. (2010). Titrating oxygen during and after cardiopulmonary resuscitation. JAMA, 303, 2190–2191.
10.
SteinT.D., AlvarezV.E., and McKeeA.C. (2014). Chronic traumatic encephalopathy: a spectrum of neuropathological changes following repetitive brain trauma in athletes and military personnel. Alzheimers Res. Ther., 6, 4
11.
GandyS., IkonomovicM.D., MitsisE., ElderG., AhlersS.T., BarthJ., StoneJ.R., and DeKoskyS.T. (2014). Chronic traumatic encephalopathy: clinical-biomarker correlations and current concepts in pathogenesis. Mol. Neurodegener., 9, 37.
12.
TitusD.J., FuronesC., AtkinsC.M., and DietrichW.D. (2015). Emergence of cognitive deficits after mild traumatic brain injury due to hyperthermia. Exp. Neurol., 263, 254–262.
13.
OjoJ.O., GreenbergM.B., LearyP., MouzonB., BachmeierC., MullanM., DiamondD.M., and CrawfordF. (2014). Neurobehavioral, neuropathological and biochemical profiles in a novel mouse model of co-morbid post-traumatic stress disorder and mild traumatic brain injury. Front. Behav. Neurosci., 8, 213.
14.
OmaluB., FitzsimmonsR.P., HammersJ., and BailesJ. (2010). Chronic traumatic encephalopathy in a professional American wrestler. J. Forensic Nurs., 6, 130–136.
15.
GoodmanM.D., MakleyA.T., HuberN.L., ClarkeC.N., FriendL.A., SchusterR.M., BaileyS.R., BarnesS.L., DorlacW.C., JohannigmanJ.A., LentschA.B., and PrittsT.A. (2011). Hypobaric hypoxia exacerbates the neuroinflammatory response to traumatic brain injury. J. Surg. Res., 165, 30–37.
