GladwellM. John Rock's errorNew Yorker March 132000; 52–63.
2.
SulakPJ. Continuous oral contraception: Changing times. Best Pract Res Clin Obstet Gynaecol, 2008; 22:355–374.
3.
VandeverMA, KuehlTJ, SulakPJet al.Evaluation of pituitary-ovarian axis suppression with three oral contraceptive regimens. Contraception, 2008; 77:162–170.
4.
SperoffL. Clinical gynecologic endocrinology and infertility, 6th. Philadelphia: Lippincott, 1999.
5.
BurkmanR, BellC, SerfatyD. The evolution of combined oral contraception: Improving the risk-to-benefit ratio. Contraception, 2011; 84:19–34.
6.
GrimesDA, GodwinAJ, RubinA, SmithJA, LacarraM. Ovulation and follicular development associated with three low-dose oral contraceptives: A randomized controlled trial. Obstet Gynecol, 1994; 83:29–34.
7.
van HeusdenAM, FauserBC. Activity of the pituitary-ovarian axis in the pill-free interval during use of low-dose combined oral contraceptives. Contraception, 1999; 59:237–243.
BäckströmT, Hansson-MalmströmY, LindheB-å, Cavalli-BjörkmanB, NordenströmS. Oral contraceptives in premenstrual syndrome: A randomized comparison of triphasic and monophasic preparations. Contraception, 1992; 46:253–268.
10.
GrahamCA, SherwinBB. A prospective treatment study of premenstrual symptoms using a triphasic oral contraceptive. J Psychosom Res, 1992; 36:257–266.
11.
JoffeH, CohenLS, HarlowBL. Impact of oral contraceptive pill use on premenstrual mood: Predictors of improvement and deterioration. Am J Obstet Gynecol, 2003; 189:1523–1530.
12.
WillisSA, KuehlTJ, SpiekermanAM, SulakPJ. Greater inhibition of the pituitary-ovarian axis in oral contraceptive regimens with a shortened hormone-free interval. Contraception, 2006; 74:100–103.