Response to Berger et al. re: “Are Pre-Ascent Low-Altitude Saliva Cortisol Levels Related to the Subsequent Acute Mountain Sickness Score? Observations From a Field Study”
Restricted accessLetterFirst published online December, 2020
Response to Berger et al. re: “Are Pre-Ascent Low-Altitude Saliva Cortisol Levels Related to the Subsequent Acute Mountain Sickness Score? Observations From a Field Study”
BergerMM, SchieferLM, SarebanM, and BärtschP. (2020). Re: “Are Pre-Ascent Low-Altitude Saliva Cortisol Levels Related to the Subsequent Acute Mountain Sickness Score? Observations From a Field Study” by Gatterer et al. (High Alt Med Biol 2019;20:337–343). High Alt Med Biol. DOI: 10.1089/ham.2020.0050
2.
ChenSJ, YangJF, KongFP, RenJL, HaoK, LiM, YuanY, ChenXC, YuRS, LiJF, LengG, ChenX-Q, and DuJ-Z. (2014). Overactivation of corticotropin-releasing factor receptor type 1 and aquaporin-4 by hypoxia induces cerebral edema. Proc Natl Acad Sci U S A, 111:13199–13204.
3.
EstoppeyJ, LegerB, VuistinerP, SartoriC, and KayserB. (2019). Low- and high-altitude cortisol awakening responses differ between AMS-prone and AMS-resistant mountaineers. High Alt Med Biol, 20:344–351.
4.
FrayserR, RennieID, GrayGW, and HoustonCS. (1975). Hormonal and electrolyte response to exposure to 17,500 ft. J Appl Physiol, 38:636–642.
5.
GattererH, BernatzkyG, BurtscherJ, RainerM, KayserB, and BurtscherM. (2019). Are pre-ascent low-altitude saliva cortisol levels related to the subsequent acute mountain sickness score? Observations from a field study. High Alt Med Biol, 20:337–343.
6.
JoyceKE, LucasSJE, ImrayCHE, BalanosGM, and WrightAD. (2018). Advances in the available non-biological pharmacotherapy prevention and treatment of acute mountain sickness and high altitude cerebral and pulmonary oedema. Expert Opin Pharmacother, 19:1891–1902.
7.
KayserB, DumontL, LysakowskiC, CombescureC, HallerG, and TramèrMR. (2012). Reappraisal of acetazolamide for the prevention of acute mountain sickness: A systematic review and meta-analysis. High Alt Med Biol, 13:82–92.
8.
SongTT, BiYH, GaoYQ, HuangR, HaoK, XuG, TangJW, MaZQ, KongFP, CooteJH, ChenX-Q, and DuJ-Z. (2016). Systemic pro-inflammatory response facilitates the development of cerebral edema during short hypoxia. J Neuroinflammation, 13:63.
9.
YoungJA, CookDI, EvansLA, and PiraniD. (1987). Effects of ion transport inhibition on rat mandibular gland secretion. J Dent Res, 66:531–536.